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廣州健侖生物科技有限公司

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【公司名稱】 廣州健侖生物科技有限公司
【市場部】    楊永漢

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【騰訊  】 2042552662
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號二期2幢101-103室

Dickins博士說強制B-ALL 細胞重新開始它們的正常發(fā)育能夠為治療白血病提供一種新策略,。“然而B-ALL與其它癌癥相比相對容易預測，目前的治療可以持續(xù)多年而且具有重大副作用,。通過了解特殊的基因改變如何驅動B-ALL,，有可能產生行動更快且較少有副作用的特異療法。”
然而Dickins博士說腫瘤細胞中的基因缺失不是傳統(tǒng)上的藥物靶點,。“癌癥發(fā)育期間通過開發(fā)藥物恢復丟失基因的功能是非常困難的,，”Dickins博士說。“然而通過了解Pax5缺失引起白血病的機制,，我們就可以開始考慮開發(fā)藥物的方式,，能夠像恢復Pax5功能一樣的效果?；蜷_關技術用于學習Pax5也能用于了解其它癌癥中的‘腫瘤抑制’基因,。”
美國亞利桑那州費城蒂格尼提健康巴羅神經(jīng)學研究所和美國加州大學圣地亞哥分校醫(yī)學院的研究人員進行的研究顯示，人類大腦會在海馬體里存儲情景記憶,，將每一段回憶交付給*的單個細胞,，這一發(fā)現(xiàn)證實了之前神經(jīng)計算理論學家長久懷疑的問題。
這個人類神經(jīng)元顯示了針對特定刺激而作出回應的肌動蛋白的形成,。

Dr. Dickins said forcing B-ALL cells to restart their normal development could provide a new strategy for the treatment of leukemia. However, B-ALL is relatively easy to predict when compared to other cancers, and the current treatment can last many years with significant side effects. By understanding how specific genetic changes drive B-ALL, it is possible to produce actions that are faster and have fewer side effects therapy."
However, Dr. Dickins said that gene deletion in tumor cells is not a traditional drug target. "It is very difficult to recover lost genes by developing drugs during cancer development," said Dr. Dickins. "However, by understanding the mechanism by which Pax5 deficiency causes leukemia, we can begin to think about ways to develop drugs that have the same effect as Pax5 functions." Gene switch technology for studying Pax5 can also be used to understand 'tumor suppression' in other cancers gene."
A recent study by researchers at the Diaghini Institute of Health Barlow Neurology in Philadelphia, Arizona, and the University of California San Diego School of Medicine found that the human brain stores episodic memories in the hippocampus, delivering each piece of memory to a unique This finding confirms the longstanding suspicion of neuro-computing theorists.
This human neuron shows the formation of actin that responds to a specific stimulus.